SPINAL CORD INJURY [PDF]

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Idea Transcript


27/04/59

บาดเจ็บไขสันหลัง คือ การ บาดเจ็บที่เกิดขึ ้นกับส่วนของไข สันหลัง ตังแต่ ้ บริ เวณ foramen magnum จนถึงส่วนปลายคือ conus medullaris ซึง่ จะอยู่ ประมาณขอบล่างของกระดูก L1 หรื อบนกระดูก L2 รวมทัง้ ส่วนของ cauda equina ด้ วย

SPINAL CORD INJURY Rehabilitation unit, Nakornping hospital

Primary Cause of Death

การตรวจทางระบบประสาทเพื่อจาแนกความรุนแรงในผู้ป่วยบาดเจ็บไขสันหลัง American Spinal Injury Association (ASIA) guidelines

การตรวจระบบประสาทรับความรู้ สกึ  28 key dermatomes ที่ตอ้ งตรวจ

pinprick & light touch ของร่างกาย

ทัง้ 2 ซีก  คะแนน 0ไม่สามารถแยกความรู ส้ ึ กแหลมกับทูไ่ ด้  คะแนน 1 แยกแหลมกับทูไ่ ด้แต่ความรู ส้ ึ กแหลมไม่เท่ากับใบหน้า  คะแนน 2 ความรู ส้ ึ กแหลมเป็ นปกติเท่ากับใบหน้า

sacral sparing ทาโดยการ PR ถ้าผูป้ ่ วยรู ส้ ึ กถึงการสัมผัส หรื อแรงกด ถื อว่ายังมี sacral sparing

 การตรวจ

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Key Muscles 

C5-elbow flexors



C6-wrist extensors



C7-elbow extensors



C8-finger flexors (distal phalanx of 3rd finger)



T1-small finger abductors

ตรวจประเมิน UE

Key Muscles 

L2- hip flexors



L3- knee extensors



L4- ankle dorsiflexors



L5- long toe extensors



S1- ankle plantar flexors



PR for sphincter tone assessment

ตรวจประเมิน LE

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ความรุนแรงของการบาดเจ็บแบ่งตาม ASIA impairment scale (revised 2000) 

ASIA:A (complete)



ASIA:B (incomplete)



ASIA:C



ASIA:D



ASIA:E

Expected Functional Outcomes by Neurologic Level of Injury

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Rehabilitation Phase of Injury  Goal

Interdisciplinary Team physician

:

Social worker

 maximizing

physical independence  becoming independent in direction of care  Preventing secondary complications pressure ulcer joint stiffness urinary tract GI tract

Family members

Recreation/v ocational therapist

PT

Patient Psychologist

etc.

OT

Dieticians

Nurses Aids

Interdisciplinary Team physician Social worker

Interdisciplinary Team The patient and family members need to be educated aboutPTthe nature of an SCI and the patient’s prognosis and the OT uncertainty of such

Family members

Recreation/v ocational therapist

Patient Psychologist

Dieticians

Nurses Aids

Rehabilitation nurses, physician in addition Social to performing worker their standard nursing duties, provideRecreation/v ocational therapist education on prevention Patient and treatment of secondary Psychologist complications, in addition to training in bowel and bladder Dieticians management.

Family members

PT

OT

Nurses

Aids

Interdisciplinary Team

Physical Skill Training

physician Social worker

Family members

Physical and occupational therapists in the acute hospital should facilitate prevention of secondary complications such as contractures, pressure ulcers, and disuse atrophy. This is done Recreation/v ocational maintenance of joint ROM, PT through therapist splinting, positioning, and selective musclePatient strengthening.



Mobility



self-care skills



other activities of daily living (ADL)

- ROM of all joints isperformed and taught by the therapists to Psychologist OT people with SCI and their caregivers as soon as it is medically safe to do so. - Performance of an adequate daily stretching program can prevent joint contractures. Dieticians Nurses - Splinting of joints, with either Aids an off-the-shelf or a custom splint fabricated by an occupational therapist, is also often used to provide a prolonged stretch, to facilitate a functional joint position, and to prevent skin breakdown.

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Practices (PT) 

joint ROM and strength



Mat activities1





rolling



prone on elbows positioning



prone on hands positioning



supine on elbows positioning,



long sitting



short sitting



quadruped positioning,



transfer training



Transfer training 

Transfer training



complete paraplegia/lower tetraplegia

stand-pivot and sit-pivot transfers

Transfer training 

complete paraplegia/lower tetraplegia

1. Lift feet onto bed and wheel the chair forward against bed. Put on brakes. Then bend forward and lift butt forward on chair.



Transfer training 



The floor-to chair transfer

1. Sit with legs straight, Pull seat to your side opposite the wheelchair (a person's knee can also be used).

2. With hands on each chair, push up, with your head forward over knees

2. With one hand on the cushion and one on the bed, lift the body sideways onto the bed.

3. Repeated lifts and lifting of legs may be needed.

Transfer training 

The floor-to chair transfer

3. Swing onto the seat. 4. Now, with your head forward over your knees, swing body onto the wheelchair.

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Standing

Wheelchair Skills

**with caution in individuals with chronic SCI** 

complete thoracic level injuries

KAFO

Other 

Spinal Cord Injury Education



Home and Environmental Modifications



Driver Training



Vocational Training

Secondary Conditions

Pulmonary System 

Pulmonary complications are the leading causes of death for people with SCI

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THE POSITION OF THE DIAPHRAGM

Management of Pulmonary Complications 

lung expansion 

Intermittent positive pressure breathing



bilevel positive airway pressure



Continuous positive airway pressure (CPAP)

Atelectasis  pneumonia, pleural effusion, empyema

Secretion mobilization techniques  Postural drainage  Percussion  Vibration

Postural drainage

Contraindication 

Severe hemoptysis



Untreated acute condition 

severe pulmonary edema



congestive heart failure



large pleural effusion



Pulmonary embolism



pneumothorax

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Contraindication 



Precaution

Cardiovascular instability



Hemoptysis



cardiac arrhythmia



Postoperation



severe hypertension or hypotension



Geriatric



recent myocardial infarction



Malignancy



Unilateral lung abscess

Recent neurosurgery

Right and Left upper lobe

Right and Left upper lobes

Right and Left lower lobes

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Percussion

Vibration

Right and Left lower lobes

Chest percussion

Orthostatic Hypotension 





Immediately after SCI  a complete loss of sympathetic tone  neurogenic (“spinal”) shock with hypotension, bradycardia, and hypothermia the sympathetic reflex activity returns  normalization of blood pressure Supraspinal control: absent in those individuals with high-level and neurologically complete SCI 

orthostatic hypotension

Vascular System 

Deep venous thrombosis (DVT)



Pulmonary embolism

Management 

elastic stockings



abdominal binders



hydration



gradually progressive daily head-up tilt



administration of salt tablets, midodrine, orfludrocortisone.

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Autonomic Dysreflexia 

syndrome and clinical emergency that affects people with SCI usually at the T6 level or above



symptoms 

pounding headache



Systolic and diastolic hypertension



profuse sweating



cutaneous vasodilatation with flushing of the face, neck,and shoulders nasal congestion



pupillary dilatation



bradycardia

Autonomic Dysreflexia 

Triggered by a noxious stimulus below the injury level 

Distended bladder



fecal impaction



pathology of the bladder and rectum



ingrown toenails



labor and delivery



surgical procedures, orgasm



Etc.

Autonomic Dysreflexia 

Treatment of acute AD 

identification of the precipitating stimulus



sat up



Loosen constrictive clothing and garments



blood pressure monitored every 2 to 5 minutes



Evacuation of the bladder done



Resolved fecal impaction



* Local anesthetic agents should be used during any manipulations of the urinary tract or rectum*



Administered fast-acting antihypertensive agents

Bowel Management

Pathophysiology of neurogenic bowel dysfunction reflexic or UMN bowel  Upper

motor neurogenic bowel (UMNB)

Suprasacral lesion

areflexic or LMN bowel

 Lower

motor neurogenic bowel (LMNB)

Conus medullaris, Cauda equina lesion

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Upper motor neurogenic bowel

↓Colonic motility 

Constipation

↓ Ability to sense the urge 

Loss volitional control incontinence



Intact spinal reflex (sacral)



Normal or increase anal sphincter tone

Summarize 



LMNB  constipation with a high risk of frequent incontinence through a lax external sphincter mechanism UMNB constipation with fecal retention behind a spastic anal sphincter  require a chemical or mechanical trigger for defecation

Lower motor neurogenic bowel



Prolonged transit time constipation

↓ anal tone

incontinence



Anorectal reflex is absent or decrease



Anocutaneous reflex is absent or decrease

Bowel Level

Outcome

Expected Outcome

Equipment

C1–4 C5

• Total assist for digital stimulation, insertion of minienema or suppository, and perineal hygiene

• Padded reclining commode chair with head support •

C6–C7

• Some to total assist for setup and perineal hygiene

• Padded commode chair • Suppository inserter • Digital bowel stimulator • Mirror

C8 T1–T12 L1–S5

• Independent digital stimulation, suppository or minienema insertion, and perineal hygiene

• Padded commode chair

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Management Neurogenic bowel management

Goal of bowel program 1. effective and efficient colonic evacuation Bowel evacuation at a consistent time of day 2. preventing incontinence 3. preventing constipation. social continence - Predictable - Scheduled - Adequate defecation without incontinence at other time

Bowel program



Fluid



Diet



Timing



Frequency



Medication



Bowel care 

Fluid 

Must be balanced with bladder management



Adequate fluid: [40xBw]+500 cc

Procedure to periodically evacuate stool from the colon

Diet 

Adequate fiber intake (No less than 15 grams of fiber daily)  Whole

grain breads and cereals, esp.

bran  Wheat  Fruits

germ and vegetables

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Timing+Frequency

Aim Timing Frequency

• To avoid chronic colorectal overdistention

• based on personal lifestyle needs • Schedule at the same time of day • bowel routine after a meal can take advantage of the gastrocolic response

Preparation



Positioning



Checking for stool



Rectal stimulation

 

4 general categories 

Stool softener



Bulk former



Peristaltic stimulant and prokinetic agent



Contact irritant

• every day in the beginning. • later on at least once every 2 days

Scheduled Bowel care 

Medication

RECTAL STIMULATION 

Pelvic nerve mediated recto-colic reflex Caution: Autonomic Dysreflexia* (T6 and above)



Mechanical

Recognising completion Clean up

Digital stimulation



Suppositories



Manual Evacuation



Mini-enema

* typically performed by a person with an LMN bowel



Inserting a gloved



lubricated finger into the rectum

inserting a gloved







slowly rotating the finger in a circular movement until relaxation of the bowel wall is felt, flatus passes, or stool passes

lubricated finger into the rectum to break up or hook stool



pull it out



typically occurs within 1 minute



repeated every 10 minutes until



*Abdominal wall massage, starting in the right lower quadrant and progressing along the course of colon

cessation of stool flow



palpable internal sphincter closure



absence of stool results from the last two digital stimulations

Chemical

Digital Stimulation

Digital evacuation

* typically effective only for people with a UMN bowel







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pulsed water irrigation



colostomy

Bladder

Bladder Level

reflexic or UMN bladder

Outcome Expected Outcome

Equipment

C1–4 C5

• Total assist for inserting • Foley catheter or external indwelling catheter catheters (transurethral or suprapubic) • Urine drainage bags or applying an external catheter to penis

C6–C7

• Total assist for inserting indwelling catheter • Independent selfcatheterization through a continent urinary diversion

• Bimanual catheter inserter • Foley, straight, or external catheters • Urine drainage bags

T1–T12 L1–S5

• Independent intermittent catheterization

• Straight catheters

The sympathetic innervation

to the bladder and bladder neck or internal urethral sphincter,

which modulates relaxation of the body of the bladder and narrowing of the bladder neck to inhibit voiding, is provided by the hypogastric nerves, which exit from the spinal cord at segments T11-L2.

Areflexic or LMN bladder

The parasympathetic innervation to the bladder, which

contraction of the urinary bladder with opening of the bladder neck to allow modulates

voiding, is provided by the pelvic splanchnic nerves, which exit from the spinal

cord at segments S2-S4.

The somatic pudendal nerve, also originating from segments S2-S4, innervates the external urinary sphincter

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Bladder dysfunction

In spinal shock, clinical will be similar

UMN type

LMN type

Lesion above sacral center

Lesion peripheral to sacral center or complete destroys sacral center



Detrusor sphincter dyssynergia.

Characterized by 

low urinary volume



high bladder pressure



uninhibited detrusor contraction



Management of Neurogenic Bladder 

goal of management 

achieve a socially acceptable method of bladder emptying



avoiding complications

Hypotonic of detrusor



Infections

and/or sphincter



Hydronephrosis with renal failure



urinary tract stones



AD

2 possible cilinical senarios Urinary retention: sphincter + / detrusor -

May trigger autonomic dysreflexia. (if lesion above the T6 vertebrae)

Continuos incontinence sphincter - / detrusor +/-

CARE IN ACUTE PHASE



Immediately after the injury (shock phase)







best option for the long-term bladder management



physiologic advantage of allowing for regular bladder filling and emptying



requires general level care



the social acceptability of not needing a drainage appliance



Indwelling catheter



fewer complications than with other methods.

Intermittent bladder catheterization (IC) 

Intermittent bladder catheterization (IC)

total fluid intake of approximately 2000 mL/day

Reflex voiding 

option for men with UMN bladder



Contractions can be triggered by various stimulation techniques

target catheterized volume of 500 mL UMN bladder: combined with anticholinergic medications

http://www.elearnsci.org/



squeezing the penis or scrotum



tapping on the suprapubic area



A condom catheter is a tube-vented condom that depends on a watertight seal for successful use



completeness of voiding can be determined by measurement of a postvoid residual urine volume



reflex voiding  elevated voiding pressures vesicoureteral reflux, hydronephrosis, and eventual renal failure

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indwelling catheter 



reasonable option for 

tetraplegia who are unable to perform IC



men who are unable to effectively maintain an external catheter on their penis

suprapubic cystostomy 



Prostatitis



Epididymitis



hypospadias

complication 

with UTI



bladder stone formation



Epididymitis



prostatitis,



Hypospadias



bladder cancer

Other method 

Avoid IC complication

Urodynamic study

Augmentation cystoplasty

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Spasticity 

Upper motor neuron



Velocity dependent, increase muscle tone and stretch reflex



Spinal cord injury  spinal shock  flaccid  flexor spasticity  extensor spasticity

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Benefit of spasticity

Indication for treatment of spasticity



Delay muscle atrophy



Interfere ADL



Decrease risk of DVT



Interfere walking, transfer, wheelchair ambulation



Decrease osteoporosis



Sleep disturbance



Improve standing and walking



Pain



Joint stiffness

Management of spasticity

Stretching of spastic muscles



Identify and get rid of noxious stimuli



Steady static stretching



Physical therapy: prolong stretching, tilt table standing, physical modalities



ROM exercises



Proper positioning



Medications: baclofen, diazepam, Tizanidine hydrochloride



Nerve block, motor point block: phenol, alcohol, botulinum toxin



Intrathecal baclofen



Surgery: rhizotomy, myelotomy

Steady static stretching

ROM exercises

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Proper positioning: supine

Proper positioning: sitting

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